Table 2 Association of established type 1 diabetes (T1D) loci in latent autoimmune diabetes in adulthood (LADA) subjects positive for both glutamic acid decarboxylase autoantibodies and insulinoma-associated antigen-2 autoantibodies. Only T1D variants significantly associated with LADA are shown (LADA association P value), as well as signals significantly different between LADA and T1D (LADA vs. T1D P value). Significance threshold is 7.46 × 10^–4 after correcting for multiple comparison. The locus reported is the closest, well-known gene of interest to the signal (a full list of genes is provided in Additional file 1: Table S3). The risk and other alleles reported refer to the alleles in T1D, and the following allele frequencies refer to the frequency of the risk allele reported in T1D for LADA, T1D, and Bone Mineral Density in Childhood Study (BMDCS) control group. Odds ratios of the risk allele reported are derived from the BMDCS control data set (n = 1057), the Wellcome Trust Case Control Consortium T1D (n = 1990), and the constrained N LADA cases (n = 309). *Independent signals (INS signals have an r² = 0.278)

From: Relative contribution of type 1 and type 2 diabetes loci to the genetic etiology of adult-onset, non-insulin-requiring autoimmune diabetes