Table 4 Association between established type 2 diabetes (T2D) loci in latent autoimmune diabetes in adulthood (LADA) cases positive for glutamic acid decarboxylase autoantibodies (GADA) and insulinoma-associated antigen-2 autoantibodies (IA2A). T2D variants that were significantly associated in LADA cases positive for GAD and IA2 autoantibodies (n = 309) (LADA association P value) are shown, as well as signals that were significantly different between LADA and T2D cases (LADA vs. T2D P value). The significance threshold was set to P < 7.04 × 10^–4 to correct for multiple testing. The locus reported is the closest, well-known gene of interest to the signal (a full list of genes is provided in Additional file 1: Table S5). T2D risk allele frequencies reported are derived from the Bone Mineral Density in Childhood Study control data set (n = 1057), the Wellcome Trust Case Control Consortium T2D (n = 1960), and the LADA cases positive for autoantibodies GADA and IA2A (n = 309). The odds ratios for LADA are shown both GEMMA-corrected (for relatedness and batch effects) and uncorrected

From: Relative contribution of type 1 and type 2 diabetes loci to the genetic etiology of adult-onset, non-insulin-requiring autoimmune diabetes