Table 3 Association of established type 2 diabetes (T2D) loci with latent autoimmune diabetes in adulthood (LADA). Only T2D variants significantly associated with LADA after correcting for multiple comparison (P < 7.04 × 10^–4) are shown (LADA association P value), as well as variants significantly different between LADA and T2D (LADA vs. T2D P value). The locus reported is the closest, well-known gene of interest to the signal (a full list of genes are provided in Additional file 1: Table S4). The risk and other alleles reported refer to the alleles in T2D, and the following allele frequencies refer to the frequency of the risk allele reported in T2D, for LADA, T2D, and Bone Mineral Density in Childhood Study (BMDCS) control groups. Odds ratios of the risk allele reported are derived from the BMDCS control data set (n = 1057), the Wellcome Trust Case Control Consortium T2D (n = 1960), and the LADA cases (n = 978)

From: Relative contribution of type 1 and type 2 diabetes loci to the genetic etiology of adult-onset, non-insulin-requiring autoimmune diabetes